Post by Admin on Feb 10, 2016 12:11:44 GMT -5
Hope the title made you, question because that was my intent. Many years I've seen the similarities and questioned why.
This person poses the same questions with their thoughts:
www.lymeneteurope.org/forum/viewtopic.php?t=4838&start=10
-Time for a Divorce
Postby Pandora » Sat 1 Jun 2013 5:59
OK this is kinda long, but surely if your here you like the real science.
Politicians will also have to become aware
they have to think towards the long term.
This new approach to medicine may cost money
(prevention, specialised units, etc)
but in the long term it will bring in money,
it will slash spending
as we won't be faced with the ever-growing costs
of people with Alzheimer's disease being cared for in homes,
of cancer treatment, AIDS treatment,
both involving long-term chemotherapy.
-------------------------------------------
Why did the worlds leading expert of PRION/AIDS/LYME say this?
Because we have NON HIV AIDS caused by spirochetes that share their genes with whatever they need to suvive.....
IT is the real Prion Europe slaughtered 200 millions cattle for in lies of MadCow.
IT is the cause of AIDS, NOT HIV,
and it is gene sharing spriochetal prion protein that cause Morgellons in all those who suffer their syndromes, psych, and cancers in their guts, in their brains, and in their skin.
With whatever junk genes it wants to share with, AND why Autism kids develop their syndromes when JUNK DNA in vaccines and GMO Gluten recombine in spirochetal prion proteins in the real seronegative AIDS. And why they cannot tolerate eating their crap junk genes.
====
HIV gp120 and OspA are the same thing, and you can't make a vaccine out of it. They failed -- all of them. Thailand with their est. 30K. reporting an AIDS LIKE illness, and countless other failures they stole our money for.
There does not seem to be any other toxin that does what Pam3Cys does, and we find it as the main antigens in HIV and Lyme.
Recently, similar desensitization experiments were reported which demonstrated that stimuli other than LPS, e.g., highly purified lipoteichoic acid (LTA) (22)
and macrophage-activating lipopeptide 2 (MALP-2) from mycoplasma (36),
can also render macrophages tolerant
to subsequent restimulation."
www.ncbi.nlm.nih.gov/pmc/articles/PMC162029/
======
YES WE TOLERATE the infections in lies of syndromes and cancers to kill us.
Generating extracellular amyloid aggregates using E. coli cells.
www.ncbi.nlm.nih.gov/pubmed/23166018
Diverse proteins
are known to be capable of
forming amyloid aggregates,
self-seeding fibrillar assemblies
that may be biologically functional
or pathological.
Well-known examples include neurodegenerative disease-associated proteins that misfold as amyloid, fungal prion proteins that can transition to a self-propagating amyloid form
and certain bacterial proteins
that fold as amyloid at the cell surface
and promote biofilm formation.
To further explore the diversity of amyloidogenic proteins, generally applicable methods for identifying them are critical.
Here we describe a cell-based method for generating amyloid aggregates that relies
on the
natural ability
of Escherichia coli cells to elaborate amyloid fibrils at the cell surface.
We use several different yeast prion proteins and the human huntingtin protein to show that protein secretion
via this specialized export pathway
promotes acquisition of the amyloid fold
specifically for proteins that have an inherent amyloid-forming propensity.
Furthermore, our findings establish the potential of this E. coli-based system
to facilitate the implementation of high-throughput screens for identifying amyloidogenic proteins and modulators of amyloid aggregation.
----------------
and the misfolded proteins with conformations
different from the normal proteins
have been proved to be the main cause for protein aggregation.
-----------------------------------
So why haven't they found out what it takes? I mean come on...Mad Cow was soooo 80's? They now propagate Prion protein where it comes from---HeLa cells and Ecoli. Surprised?
tgs.freshpatents.com/Prion-bx1.php
patents.com/search/?top_keyword=Prion&sa=Search
prionpatents.com/index.html
One of the same reasons we are dying....
Imagine our gene columns stripped of their protection mechanisms of our TLR's that vaccines turned off...Introduce genetically altered organisms DNA that glob around those gene columns and pick and choose what they need from those globs to survive.
That is the real AIDS/PRION/LYME killing the public.
---
www.ncbi.nlm.nih.gov/pubmed/23614720
However, recent exciting data
suggest that the transmissibility of misfolded proteins within the brain
is a property that goes """way beyond the rare prion diseases."""
-------------
How many infections do you suppose she suffers?
... playnext=1
-------------
Probable spirochetal etiology could be demonstrated for 41 of 45 (91%) patients with clinical symptoms of chronic meningitis.
Approximately 25% of the patients had significantly elevated titers of antibody to the spirochete in CSF but not in serum.
www.researchgate.net/publication ... ub_cit_inc
----------
So if they found spirochetal disease in 41 of 45 people with Meningitis what exactly is it they are vaccinating to prevent? Hey folks your paying for it don't you think you ought to know what they are giving your kids? OSPA perhaps?
Weak tendons, ligaments, bones are caused by infections they refuse to treat. And too stupid to culture.
www.ncbi.nlm.nih.gov/pubmed?term ... eoscarcoma
www.ncbi.nlm.nih.gov/pubmed/23614720
However, recent exciting data suggest that the transmissibility of misfolded proteins within the brain is a property that goes """"way beyond the rare prion diseases.""""
synuclein-gamma
antibodies.cancer.gov/apps/site/ ... PTC-SNCG-1
It now appears that lymphotoxin may bridge the gap
between altered composition
of the commensal microbiota and metabolism.
www.ncbi.nlm.nih.gov/pubmed/23657002
LOL....
www.ncbi.nlm.nih.gov/pubmed/22912582
Lymphotoxin, but not TNF, is required for prion invasion of lymph nodes.
---
Of course no one makes them detect or culture spirochetes...
PEOPLE 1/3 TO 1/2 OF THE POP.S HAVE A "LATENT" FORM OF TB---90% OF THE POPS HAVE EBV AND OTHER HERPES SIMILARS....1/3 are found to have Toxoplasmosis, and I am sure when someone bothers to look like France, Russia and Italy have have they will find all the infections too.
YOU DO "NOT" NEED TO ASK WHAT VACCINES HAVE PROTECTED YOU FROM ANYMORE----ABSOLUTELY NOTHING.
THEY HAVE DESTROYED MAN AND BEAST ALIKE TO THE EDGE OF THE ABYSS.....
Recently, similar desensitization experiments were reported which demonstrated that stimuli other than LPS, e.g., highly purified lipoteichoic acid (LTA) (22)
and macrophage-activating lipopeptide 2 (MALP-2) from mycoplasma (36),
can also render macrophages
tolerant
to subsequent restimulation."
www.ncbi.nlm.nih.gov/pmc/articles/PMC162029/
YES WE TOLERATE the infections in lies of syndromes and cancers to kill us.
Generating extracellular amyloid aggregates using E. coli cells.
www.ncbi.nlm.nih.gov/pubmed/23166018
Diverse proteins are known to be capable of
forming amyloid aggregates,
self-seeding
fibrillar assemblies
that may be biologically functional
or pathological.
--
Well-known examples include neurodegenerative disease-associated proteins that misfold as amyloid, fungal prion proteins that can transition to a self-propagating amyloid form
and certain bacterial proteins
that fold as amyloid at the cell surface
and promote biofilm formation.
To further explore the diversity of amyloidogenic proteins, generally applicable methods for identifying them are critical.
Here we describe a cell-based method for generating amyloid aggregates that relies
on the natural ability
of Escherichia coli cells to elaborate
amyloid fibrils at the cell surface.
We use several different yeast prion proteins and the human huntingtin protein to show that protein secretion
via this specialized export pathway
promotes acquisition of the amyloid fold
specifically for proteins that have an inherent amyloid-forming propensity.
Furthermore, our findings establish the potential of this E. coli-based system
to facilitate the implementation of high-throughput screens for identifying amyloidogenic proteins and modulators of amyloid aggregation.
----------------
Imagine our gene columns stripped of their protection mechanisms of our TLR's that vaccines turned off...Introduce genetically altered organisms that glob around those gene columns and pick and choose what they need from those globs to survive.
That is the real AIDS/PRION/LYME killing the public.
newjournal.kcsnet.or.kr/main/j_s ... s&dpage=ar
Conventional analytical procedures (e.g., Edman degradation and amino acid analysis)
are either not applicable
due to the N-terminal modification,
or do not provide confirmation of the intact structure.
Chromatographic analysis is also hindered by the tendency of these
lipoidal Pam3Cys peptides to form large aggregates,
and in some cases to be permanently adsorbed
on reversed phase columns.
We have applied several mass spectrometric techniques,
including fast atom bombardment (FAB), electrospray ionization (ESI) and matrix assisted laser desorption ionization (MALDI) to characterize the intact structures of a number of different Pam3Cys synthetic peptides.
1996=Totally ignored to protect profits. And they knew the same back in 1985 and did nothing. The infections cause cancers and all syndromes.
www.ncbi.nlm.nih.gov/pubmed/23689138
LMP1 is secreted via exosomes, is incorporated into EBV-uninfected cells by endocytosis, and affects the environment surrounding the tumor. Here we reviewed the contribution of EBV gene products to NPC pathogenesis in relation with LMP1.
It is also the cause of Morgellons in the masses...only they refuse to look or produce an exit so you can see just how badly the public is infected..Its quite easy to see with a micro magnifying glass after first enducing an exit with animal wormer paste or some mild drops of acid like lemon.
www.morgellons-research.org/morg ... oscop3.htm
So why didn't they tell you it attaches junk DNA to your gene columns when injected?
www.ncbi.nlm.nih.gov/pu...
arxiv.org/ftp/arxiv/papers/1301/1301.2845.pdf
www.ncbi.nlm.nih.gov/pu...
www.morgellons-research.org/morg ... oscop3.htm
f1000research.com/articles/2-25/v1
Proving
Lipid Rafts Exist: Membrane Domains in the Prokaryote Borrelia
burgdorferi Have the Same Properties as Eukaryotic Lipid Rafts.
www.ncbi.nlm.nih.gov/pubmed/?ter ... 20Prokaryo
[Infectious properties of protein aggregates involved in neurodegenerative diseases].
www.ncbi.nlm.nih.gov/pubmed/?term=[Infectious%20properties%20of%20protein%20aggregates%20involved%20in%20neurodegenerative%20diseases].
www.ncbi.nlm.nih.gov/pm...
The plasmids together function as a portable incubator for genetic
innovation that allows Bb to accomplish immune evasion feats not
witnessed elsewhere in the bacterial kingdom.
That's why the ARMY classified them as bioweapons and played with spirochetes
for 50yrs. before deciding to use them in vaccines.
www.ncbi.nlm.nih.gov/pubmed/23678876
These phenotypes can be so distinctive that they would seem to have differing
biology. However,
they cannot be distinguished, at least
neuropathologically or genetically.
In sporadic ALS (SALS), they are
mostly characterized by ubiquitinated
cytoplasmic inclusions of TDP-43.
In familial ALS (FALS), where phenotypes are indistinguishable from SALS
and similarly heterogeneous,
each mutated gene has its own genetic and
molecular signature.
------------------------
Hey,,,that ain't HIV he's talkin about.
Neither was this guy....
abclocal.go.com/wtvd/video?id=91 ... id=9121538
Why? BECAUSE THERE IS NO MORE NORMAL. WE ARE ALL INFECTED WITH STEALTH.
The amyloid-forming proteins tau, αB crystallin, and amyloid P protein
are all found in lesions of multiple sclerosis (MS).
Our previous work established that amyloidogenic peptides from the small heat shock protein αB crystallin (HspB5)
and from amyloid β fibrils,
characteristic of Alzheimer's disease,
www.ncbi.nlm.nih.gov/pu...
How long have the skum bags known and not helped the people?
The
newly synthesized DNA in IFN-beta-treated cells was replicative and not
repair DNA. The observation that IFN-beta inhibits the processing of
newly synthesized low molecular weight DNA into normal DNA might be
explained by the intracellular accumulation of S-adenosylhomocysteine in
IFN-beta-treated HeLa cells (de Ferra, F., and Baglioni, C.
(1983) J.
Biol. Chem. 258, 2118-2121) which could change the soluble
ribonucleotide and deoxyribonucleotide pool and ultimately affect DNA
processing.
www.jbc.org/content/259...
Now they splain it all away by telling you to just be Happy!!! LOL
www.whydontyoutrythis.com/2013/0 ... WG4cc.dpbs
That is just a drop of the science proving their crimes against humanity. We only went to war to take out USA with more junk to recombine in the spirochetal prion protein infections we already have. Why else would thousands suffer lies of PTSD, and 22 a day keel themselves a day denied treatment for the infections?
They knew in 1956 what they were giving us.
Do you know how to give Bee's their own stem cells because they and the bats are infected from using contaminated feces for fertilizers and bats and birds like martins eating blood eating moquito's ?
No worries. Monstersanto stole all the NON GMO Bee's they could muster.
Now take your 5 doses of "ETHICALLY" approved Anthrax vaccine for your sick kiddies and lets get this shoe on the road..
HeLa cells for vaccine use
You may have all the faith in the world in pharma after letting millions suffer lies of Autism caused by infectious JUNK DNA in vaccines recombining in spirochetal disease they KNEW could not be killed===Until its one of YOURS....
... playnext=1
We humans were never intended to have spirochetal Junk in vaccines, or Ecoli God made to take care of fecal waste injected into our skin. They have destroyed our immune systems, they have stolen billions of live in lies, and stolen countless Tax dollars, not to mention how much their criminal lies have cost pts.
Its not only time to DIVORCE LYME, Its time to DIVORCE the whole Medical Mafia lies of protections, lies of meds used just to treat the symptoms, lies of who and why we suffer spirochetal disease.
This person poses the same questions with their thoughts:
www.lymeneteurope.org/forum/viewtopic.php?t=4838&start=10
-Time for a Divorce
Postby Pandora » Sat 1 Jun 2013 5:59
OK this is kinda long, but surely if your here you like the real science.
Politicians will also have to become aware
they have to think towards the long term.
This new approach to medicine may cost money
(prevention, specialised units, etc)
but in the long term it will bring in money,
it will slash spending
as we won't be faced with the ever-growing costs
of people with Alzheimer's disease being cared for in homes,
of cancer treatment, AIDS treatment,
both involving long-term chemotherapy.
-------------------------------------------
Why did the worlds leading expert of PRION/AIDS/LYME say this?
Because we have NON HIV AIDS caused by spirochetes that share their genes with whatever they need to suvive.....
IT is the real Prion Europe slaughtered 200 millions cattle for in lies of MadCow.
IT is the cause of AIDS, NOT HIV,
and it is gene sharing spriochetal prion protein that cause Morgellons in all those who suffer their syndromes, psych, and cancers in their guts, in their brains, and in their skin.
With whatever junk genes it wants to share with, AND why Autism kids develop their syndromes when JUNK DNA in vaccines and GMO Gluten recombine in spirochetal prion proteins in the real seronegative AIDS. And why they cannot tolerate eating their crap junk genes.
====
HIV gp120 and OspA are the same thing, and you can't make a vaccine out of it. They failed -- all of them. Thailand with their est. 30K. reporting an AIDS LIKE illness, and countless other failures they stole our money for.
There does not seem to be any other toxin that does what Pam3Cys does, and we find it as the main antigens in HIV and Lyme.
Recently, similar desensitization experiments were reported which demonstrated that stimuli other than LPS, e.g., highly purified lipoteichoic acid (LTA) (22)
and macrophage-activating lipopeptide 2 (MALP-2) from mycoplasma (36),
can also render macrophages tolerant
to subsequent restimulation."
www.ncbi.nlm.nih.gov/pmc/articles/PMC162029/
======
YES WE TOLERATE the infections in lies of syndromes and cancers to kill us.
Generating extracellular amyloid aggregates using E. coli cells.
www.ncbi.nlm.nih.gov/pubmed/23166018
Diverse proteins
are known to be capable of
forming amyloid aggregates,
self-seeding fibrillar assemblies
that may be biologically functional
or pathological.
Well-known examples include neurodegenerative disease-associated proteins that misfold as amyloid, fungal prion proteins that can transition to a self-propagating amyloid form
and certain bacterial proteins
that fold as amyloid at the cell surface
and promote biofilm formation.
To further explore the diversity of amyloidogenic proteins, generally applicable methods for identifying them are critical.
Here we describe a cell-based method for generating amyloid aggregates that relies
on the
natural ability
of Escherichia coli cells to elaborate amyloid fibrils at the cell surface.
We use several different yeast prion proteins and the human huntingtin protein to show that protein secretion
via this specialized export pathway
promotes acquisition of the amyloid fold
specifically for proteins that have an inherent amyloid-forming propensity.
Furthermore, our findings establish the potential of this E. coli-based system
to facilitate the implementation of high-throughput screens for identifying amyloidogenic proteins and modulators of amyloid aggregation.
----------------
and the misfolded proteins with conformations
different from the normal proteins
have been proved to be the main cause for protein aggregation.
-----------------------------------
So why haven't they found out what it takes? I mean come on...Mad Cow was soooo 80's? They now propagate Prion protein where it comes from---HeLa cells and Ecoli. Surprised?
tgs.freshpatents.com/Prion-bx1.php
patents.com/search/?top_keyword=Prion&sa=Search
prionpatents.com/index.html
One of the same reasons we are dying....
Imagine our gene columns stripped of their protection mechanisms of our TLR's that vaccines turned off...Introduce genetically altered organisms DNA that glob around those gene columns and pick and choose what they need from those globs to survive.
That is the real AIDS/PRION/LYME killing the public.
---
www.ncbi.nlm.nih.gov/pubmed/23614720
However, recent exciting data
suggest that the transmissibility of misfolded proteins within the brain
is a property that goes """way beyond the rare prion diseases."""
-------------
How many infections do you suppose she suffers?
... playnext=1
-------------
Probable spirochetal etiology could be demonstrated for 41 of 45 (91%) patients with clinical symptoms of chronic meningitis.
Approximately 25% of the patients had significantly elevated titers of antibody to the spirochete in CSF but not in serum.
www.researchgate.net/publication ... ub_cit_inc
----------
So if they found spirochetal disease in 41 of 45 people with Meningitis what exactly is it they are vaccinating to prevent? Hey folks your paying for it don't you think you ought to know what they are giving your kids? OSPA perhaps?
Weak tendons, ligaments, bones are caused by infections they refuse to treat. And too stupid to culture.
www.ncbi.nlm.nih.gov/pubmed?term ... eoscarcoma
www.ncbi.nlm.nih.gov/pubmed/23614720
However, recent exciting data suggest that the transmissibility of misfolded proteins within the brain is a property that goes """"way beyond the rare prion diseases.""""
synuclein-gamma
antibodies.cancer.gov/apps/site/ ... PTC-SNCG-1
It now appears that lymphotoxin may bridge the gap
between altered composition
of the commensal microbiota and metabolism.
www.ncbi.nlm.nih.gov/pubmed/23657002
LOL....
www.ncbi.nlm.nih.gov/pubmed/22912582
Lymphotoxin, but not TNF, is required for prion invasion of lymph nodes.
---
Of course no one makes them detect or culture spirochetes...
PEOPLE 1/3 TO 1/2 OF THE POP.S HAVE A "LATENT" FORM OF TB---90% OF THE POPS HAVE EBV AND OTHER HERPES SIMILARS....1/3 are found to have Toxoplasmosis, and I am sure when someone bothers to look like France, Russia and Italy have have they will find all the infections too.
YOU DO "NOT" NEED TO ASK WHAT VACCINES HAVE PROTECTED YOU FROM ANYMORE----ABSOLUTELY NOTHING.
THEY HAVE DESTROYED MAN AND BEAST ALIKE TO THE EDGE OF THE ABYSS.....
Recently, similar desensitization experiments were reported which demonstrated that stimuli other than LPS, e.g., highly purified lipoteichoic acid (LTA) (22)
and macrophage-activating lipopeptide 2 (MALP-2) from mycoplasma (36),
can also render macrophages
tolerant
to subsequent restimulation."
www.ncbi.nlm.nih.gov/pmc/articles/PMC162029/
YES WE TOLERATE the infections in lies of syndromes and cancers to kill us.
Generating extracellular amyloid aggregates using E. coli cells.
www.ncbi.nlm.nih.gov/pubmed/23166018
Diverse proteins are known to be capable of
forming amyloid aggregates,
self-seeding
fibrillar assemblies
that may be biologically functional
or pathological.
--
Well-known examples include neurodegenerative disease-associated proteins that misfold as amyloid, fungal prion proteins that can transition to a self-propagating amyloid form
and certain bacterial proteins
that fold as amyloid at the cell surface
and promote biofilm formation.
To further explore the diversity of amyloidogenic proteins, generally applicable methods for identifying them are critical.
Here we describe a cell-based method for generating amyloid aggregates that relies
on the natural ability
of Escherichia coli cells to elaborate
amyloid fibrils at the cell surface.
We use several different yeast prion proteins and the human huntingtin protein to show that protein secretion
via this specialized export pathway
promotes acquisition of the amyloid fold
specifically for proteins that have an inherent amyloid-forming propensity.
Furthermore, our findings establish the potential of this E. coli-based system
to facilitate the implementation of high-throughput screens for identifying amyloidogenic proteins and modulators of amyloid aggregation.
----------------
Imagine our gene columns stripped of their protection mechanisms of our TLR's that vaccines turned off...Introduce genetically altered organisms that glob around those gene columns and pick and choose what they need from those globs to survive.
That is the real AIDS/PRION/LYME killing the public.
newjournal.kcsnet.or.kr/main/j_s ... s&dpage=ar
Conventional analytical procedures (e.g., Edman degradation and amino acid analysis)
are either not applicable
due to the N-terminal modification,
or do not provide confirmation of the intact structure.
Chromatographic analysis is also hindered by the tendency of these
lipoidal Pam3Cys peptides to form large aggregates,
and in some cases to be permanently adsorbed
on reversed phase columns.
We have applied several mass spectrometric techniques,
including fast atom bombardment (FAB), electrospray ionization (ESI) and matrix assisted laser desorption ionization (MALDI) to characterize the intact structures of a number of different Pam3Cys synthetic peptides.
1996=Totally ignored to protect profits. And they knew the same back in 1985 and did nothing. The infections cause cancers and all syndromes.
www.ncbi.nlm.nih.gov/pubmed/23689138
LMP1 is secreted via exosomes, is incorporated into EBV-uninfected cells by endocytosis, and affects the environment surrounding the tumor. Here we reviewed the contribution of EBV gene products to NPC pathogenesis in relation with LMP1.
It is also the cause of Morgellons in the masses...only they refuse to look or produce an exit so you can see just how badly the public is infected..Its quite easy to see with a micro magnifying glass after first enducing an exit with animal wormer paste or some mild drops of acid like lemon.
www.morgellons-research.org/morg ... oscop3.htm
So why didn't they tell you it attaches junk DNA to your gene columns when injected?
www.ncbi.nlm.nih.gov/pu...
arxiv.org/ftp/arxiv/papers/1301/1301.2845.pdf
www.ncbi.nlm.nih.gov/pu...
www.morgellons-research.org/morg ... oscop3.htm
f1000research.com/articles/2-25/v1
Proving
Lipid Rafts Exist: Membrane Domains in the Prokaryote Borrelia
burgdorferi Have the Same Properties as Eukaryotic Lipid Rafts.
www.ncbi.nlm.nih.gov/pubmed/?ter ... 20Prokaryo
[Infectious properties of protein aggregates involved in neurodegenerative diseases].
www.ncbi.nlm.nih.gov/pubmed/?term=[Infectious%20properties%20of%20protein%20aggregates%20involved%20in%20neurodegenerative%20diseases].
www.ncbi.nlm.nih.gov/pm...
The plasmids together function as a portable incubator for genetic
innovation that allows Bb to accomplish immune evasion feats not
witnessed elsewhere in the bacterial kingdom.
That's why the ARMY classified them as bioweapons and played with spirochetes
for 50yrs. before deciding to use them in vaccines.
www.ncbi.nlm.nih.gov/pubmed/23678876
These phenotypes can be so distinctive that they would seem to have differing
biology. However,
they cannot be distinguished, at least
neuropathologically or genetically.
In sporadic ALS (SALS), they are
mostly characterized by ubiquitinated
cytoplasmic inclusions of TDP-43.
In familial ALS (FALS), where phenotypes are indistinguishable from SALS
and similarly heterogeneous,
each mutated gene has its own genetic and
molecular signature.
------------------------
Hey,,,that ain't HIV he's talkin about.
Neither was this guy....
abclocal.go.com/wtvd/video?id=91 ... id=9121538
Why? BECAUSE THERE IS NO MORE NORMAL. WE ARE ALL INFECTED WITH STEALTH.
The amyloid-forming proteins tau, αB crystallin, and amyloid P protein
are all found in lesions of multiple sclerosis (MS).
Our previous work established that amyloidogenic peptides from the small heat shock protein αB crystallin (HspB5)
and from amyloid β fibrils,
characteristic of Alzheimer's disease,
www.ncbi.nlm.nih.gov/pu...
How long have the skum bags known and not helped the people?
The
newly synthesized DNA in IFN-beta-treated cells was replicative and not
repair DNA. The observation that IFN-beta inhibits the processing of
newly synthesized low molecular weight DNA into normal DNA might be
explained by the intracellular accumulation of S-adenosylhomocysteine in
IFN-beta-treated HeLa cells (de Ferra, F., and Baglioni, C.
(1983) J.
Biol. Chem. 258, 2118-2121) which could change the soluble
ribonucleotide and deoxyribonucleotide pool and ultimately affect DNA
processing.
www.jbc.org/content/259...
Now they splain it all away by telling you to just be Happy!!! LOL
www.whydontyoutrythis.com/2013/0 ... WG4cc.dpbs
That is just a drop of the science proving their crimes against humanity. We only went to war to take out USA with more junk to recombine in the spirochetal prion protein infections we already have. Why else would thousands suffer lies of PTSD, and 22 a day keel themselves a day denied treatment for the infections?
They knew in 1956 what they were giving us.
Do you know how to give Bee's their own stem cells because they and the bats are infected from using contaminated feces for fertilizers and bats and birds like martins eating blood eating moquito's ?
No worries. Monstersanto stole all the NON GMO Bee's they could muster.
Now take your 5 doses of "ETHICALLY" approved Anthrax vaccine for your sick kiddies and lets get this shoe on the road..
HeLa cells for vaccine use
You may have all the faith in the world in pharma after letting millions suffer lies of Autism caused by infectious JUNK DNA in vaccines recombining in spirochetal disease they KNEW could not be killed===Until its one of YOURS....
... playnext=1
We humans were never intended to have spirochetal Junk in vaccines, or Ecoli God made to take care of fecal waste injected into our skin. They have destroyed our immune systems, they have stolen billions of live in lies, and stolen countless Tax dollars, not to mention how much their criminal lies have cost pts.
Its not only time to DIVORCE LYME, Its time to DIVORCE the whole Medical Mafia lies of protections, lies of meds used just to treat the symptoms, lies of who and why we suffer spirochetal disease.